Universität zu Köln

Franz, Andre and Hoppe, Thorsten ORCID: 0000-0002-4734-9352 (2018). Stressed Out: Mitohormesis Is Crossing Borders. Trends Endocrinol. Metab., 29 (8). S. 525 - 528. LONDON: ELSEVIER SCIENCE LONDON. ISSN 1879-3061

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Abstract

Labbadia et al. showed that low-dose mitochondrial stress promotes protein homeostasis in the cytosol to endure proteotoxic conditions, particularly during aging. This hormetic mitochondrial stress response is heat shock factor 1 (HSF1)-dependent and, remarkably, does not affect physiological parameters that are usually associated with pathogenic disturbance of mitochondrial function.

Item Type:	Journal Article
Creators:	Creators Email ORCID ORCID Put Code Franz, Andre UNSPECIFIED UNSPECIFIED UNSPECIFIED Hoppe, Thorsten UNSPECIFIED orcid.org/0000-0002-4734-9352 UNSPECIFIED
URN:	urn:nbn:de:hbz:38-179002
DOI:	10.1016/j.tem.2018.03.007
Journal or Publication Title:	Trends Endocrinol. Metab.
Volume:	29
Number:	8
Page Range:	S. 525 - 528
Date:	2018
Publisher:	ELSEVIER SCIENCE LONDON
Place of Publication:	LONDON
ISSN:	1879-3061
Language:	English
Faculty:	Faculty of Mathematics and Natural Sciences
Divisions:	Faculty of Mathematics and Natural Sciences > Department of Biology > Institute for Genetics
Subjects:	no entry
Uncontrolled Keywords:	Keywords Language MITOCHONDRIAL; LONGEVITY; PROTEOSTASIS; SIGNALS Multiple languages Endocrinology & Metabolism Multiple languages
Refereed:	Yes
URI:	http://kups.ub.uni-koeln.de/id/eprint/17900