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Journal Article
Andersson, E. I., Puetzer, S., Yadav, B., Dufva, O., Khan, S., He, L., Sellner, L., Schrader, A., Crispatzu, G., Oles, M., Zhang, H., Adnan-Awad, S., Lagstrom, S., Bellanger, D., Mpindi, J. P., Eldfors, S., Pemovska, T., Pietarinen, P., Lauhio, A., Tomska, K., Cuesta-Mateos, C., Faber, E., Koschmieder, S., Bruemmendorf, T. H., Kytola, S., Savolainen, E-R, Siitonen, T., Ellonen, P., Kallioniemi, O., Wennerberg, K., Ding, W., Stern, M-H, Huber, W., Anders, S., Tang, J., Aittokallio, T., Zenz, T., Herling, M. and Mustjoki, S. (2018). Discovery of novel drug sensitivities in T-PLL by high-throughput ex vivo drug testing and mutation profiling. Leukemia, 32 (3). S. 774 - 788. LONDON: NATURE PUBLISHING GROUP. ISSN 1476-5551
Coppe, A., Andersson, E. I., Binatti, A., Gasparini, V. R., Bortoluzzi, S., Clemente, M., Herling, M., Maciejewski, J., Mustjoki, S. and Bortoluzzi, S. (2017). Genomic landscape characterization of large granular lymphocyte leukemia with a systems genetics approach. Leukemia, 31 (5). S. 1243 - 1248. LONDON: NATURE PUBLISHING GROUP. ISSN 1476-5551
Puetzer, S., Varghese, L., von Jan, J., Braun, T., Giri, A. K., Mayer, P., Riet, N., Timonen, S., Oberbeck, S., Kuusanmaki, H., Mustjoki, S., Stern, M. -H., Aittokallio, T., Newrzela, S., Schrader, A. and Herling, M. (2020). Reinstated p53 response and high anti-T-cell leukemia activity by the novel alkylating deacetylase inhibitor tinostamustine. Leukemia, 34 (9). S. 2513 - 2519. LONDON: NATURE PUBLISHING GROUP. ISSN 1476-5551
Schrader, A., Crispatzu, G., Oberbeck, S., Mayer, P., Putzer, S., von Jan, J., Vasyutina, E., Warner, K., Weit, N., Pflug, N., Braun, T., Andersson, E. I., Yadav, B., Riabinska, A., Maurer, B., Ferreira, M. S. Ventura, Beier, F., Altmueller, J., Lanasa, M., Herling, C. D., Haferlach, T., Stilgenbauer, S., Hopfinger, G., Peifer, M., Bruemmendorf, T. H., Nuernberg, P., Elenitoba-Johnson, K. S. J., Zha, S., Hallek, M., Moriggl, R., Reinhardt, H. C., Stern, M. -H., Mustjoki, S., Newrzela, S., Frommolt, P. and Herling, M. (2018). Actionable perturbations of damage responses by TCL1/ATM and epigenetic lesions form the basis of T-PLL. Nat. Commun., 9. LONDON: NATURE PUBLISHING GROUP. ISSN 2041-1723